Now, even if pot wasn't the safest of all recreational drugs, even if it were the deadliest, how does your neighbor getting stoned affect you or society at large?
I believe I've said this before, but I'll rephrase it. If my neighbor wants to get stoned at home and stay home, I don't care. Hell he can do heroin, coke, whatever he wants at home as long as he stays home. I don't care if he uses a combination of different recreational drugs as long as he stays home.
I have a problem when people who are under the influence go out and interfere with non-intoxicated society. If someone has too much to drink and goes out wandering around they can expect to be charged with drunk in public, if they are stupid enough to attempt to drive a car they get DUI. I want to see the same for all recreational drugs. At that point, we would truly be treating them all the same.
The Pro Pot Propaganda Pushers should support this if they really want pot treated the same as alcohol. Instead we have many people who want pot treated more like water than like alcohol.
I believe I have also stated before that what I want to see is a reliable and quantifiable test for when someone has had too much to be out in public. Again, this is what we do with alcohol - I want to see it for any recreational drug that we legalize for general consumption.
Because I feel like it. I need no reason, or purpose.
True, you need no purpose. I don't care that you have none, but the lack of a purpose does make you look rather ridiculous.
And I don't need to back up anything to you.
True, but by never backing anything up you end up supporting the hypothesis that nothing you write comes with any sincerity at all
You are free to ignore. I do not care.
Considering how often you write replies to what I write - particularly since you reply to what I write without reading what I write - you can't make much of an argument for not caring.
Except I do vote for living breathing people
You say that, but have never given any reason for me to believe it. I have asked you many times to name a candidate who you support, and not once have you ever named one for any race. You have repeatedly claimed to know better than I a candidate who would support what would be best for me, yet that person has never been given a name.
Frankly, I highly doubt you vote at all. I expect most likely you just jerk off to CSPAN on election day and then tell everyone else the day after how wrong they were for voting when they could have been pleasuring themselves at home all day instead.
The assessment of toxicological endpoints and BMD for the selected known and suspected human carcinogens was generally based on literature data, as own doseâ"response modeling would have gone beyond the scope of our study. Suitable risk assessment studies including endpoints and doseâ"response modeling results were typically identified in monographs of national and international risk assessments bodies such as WHO IPCS, JECFA, US EPA and EFSA. For substances without available monographs or with missing data on doseâ"response modeling results, the scientific literature in general was searched for such data. Searches were carried out in September 2011 in the following databases: PubMed (US National Library of Medicine, Bethesda, MD), Web of Science (Thomson Reuters, Philadelphia, PA), Scopus (Elsevier B.V., Amsterdam, The Netherlands) and Google Scholar (Google, Mountain View, CA).
The BMD/MOE approach was used for risk assessment.13, 14 In short, the BMD is the dose of a substance that produces a predetermined change in response rate (benchmark response) of an adverse effect compared to background based on doseâ"response modeling.14 The benchmark response is generally set near the lower limit of responses that can be measured (typically in the range of 1â"10%). The result of BMD-response modeling can then be used in combination with exposure data to calculate a MOE for quantitative risk assessment. The MOE is defined as the ratio between the lower one-sided confidence limit of the BMD (BMDL) and estimated human intake of the same compound. It can be used to compare the health risk of different compounds and in turn prioritize risk management actions. By definition, the lower the MOE, the larger the risk for humans; generally, a value under 10,000 used to define public health risks.15
So really, this is about the overall health risks of a substance. Certainly important but that is far from being an endorsement of any of the substances for routine use.
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