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Submission + - AI Needs Infrastructure — But Does It Need Us? (travelwatchnews.com)

gethotelreviews writes: Artificial intelligence may be digital, but its existence isn't. It requires electricity, chips, cooling, data centres, communications networks and physical machinery.

Today humans build and maintain that infrastructure. At what point could AI maintain the systems upon which its own intelligence depends?

ReadWhat Happens to Travel in an AI World? Travel Watch New

Comment We're probably going to find more (Score 4, Interesting) 34

Its only a matter of time before we find more cancers attributable to infections. Classically, it was anorectal, cervical, laryngeal and oropharyngeal squamous cell carcinomata that were attributed to HPV. It'll be a matter of time before we identify the serovar of HPV that results in cutaneous SCC - and I suspect, in my lifetime, we will see a reduction in cutaneous SCC cases due to vaccination with Gardasil. https://doi.org/10.3390/vaccin...

Comment WHO data? (Score 1, Troll) 245

I wonder if the esteemed doctor would have come to the same conclusion had the data been obtained primarily ie. by his own team, rather than derived from secondary analysis of WHO data. Recall that the Aylward WHO mission to Wuhan to study this was obstructed and delayed and didnâ(TM)t take place until mid-late February 2020, and Li-Wenliang sounded the alarm on this in Dec 30. Regardless of whether it originated in a market bat and simply spread as Coronaviridae spp do, or a population was deliberately inoculated from a laboratory derived source, it does not diminish the fact that the save-face culture that CCP administration professed has killed thousands of its own citizens by late action.

Comment Re: Begs the question... (Score 5, Informative) 185

Crossreactivity of antibodies between different viral species of the same genus is a recognised phenomenon. Vaccinia infections (cowpox) to protect against variola (smallpox) is THE classic example - the same antibodies that killed cowpox also killed smallpox. We might be seeing a similar phenomenon here with a related non-SARS, non-COVID coronavirus. The thing about COVID-19 is so few actually develop functioning antibodies to the virus. The fact that it infects T-helper cells has something to do with this, I believe.

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