Depends on the Building in question. For instance, night time photos of the Eiffel Tower are under copyright. From Wikipedia:

Really? I needed a couple more points anyway.

Had a similar experience back in college. I was playing about 2 hours a day of UT2k4 with instagib activated and fencing (the kind with swords) for 4 hours a week. My reflexes were noticeably faster than my compatriots, and I had a much easier time picking out and recognizing small details. The place that it was most apparent was actually in my entomology class when we had to go out and collect insects. I had an easier time acquiring specimens simply because I noticed them when others didn't. The faster reflexes also helped me catch some of the more evasive ones. So yeah, if ya ever take an entomology class, the videogames do help.

I'm not a virologist either, but I have taken classes in the subject (some time ago, so don't take what I say as doctrine or anything). The idea of a competing virus does have some merit. IIRC, research was done on the topic with ex vivo models, but I don't think it ever made its way to animal subject or human trials.

For this to work, you'd ideally want a virus which used the same antigen for cellular entry (gp120, CD40 ligand); this keeps your virus to the same cells that are susceptible to HIV, limiting your spread within the organism. As for an anti-HIV payload, you'd probably want to try siRNA transcripts targeted against HIV's Reverse Transcriptase or coat proteins. It would be fairly trivial to engineer your own virus to avoid the siRNAs. Select your upstream promoters very carefully and you can control the spread of your engineered virus to some extent.

What you end up getting is a way to regulate how much HIV spreads within the organism. This *would not* prevent infection with HIV, but it theoretically *could* prevent AIDS. Making it work really depends on the specificity of the siRNAs and the choice of promoters/regulatory elements in your virus' design (antibody-activated promoters are a good idea).

But like I said, it's been a while since my virology and recombinant DNA tech classes, so I'm sure there's plenty of necessary details that I'm leaving out.

The chance of it surviving in the wild is a fairly remote possibility. All the extra genes and modified genes create an extra metabolic payload that puts this strain at a disadvantage compared to the wild strains. It is possible that the sugars liberated by these extra enzymes may be enough to overcome the difference, but it's unlikely. Additionally, (and I haven't RTFA) normally when genes are modified/added to an organism, the vector that carries them also carries the genetic switch to turn certain genes on and off. For instance, it may only produce the worrisome enzyme when in the presence of a certain antibiotic like ampicillin. In wild conditions such a trigger would be absent, and the enzymes wouldn't be produced anyway. Furthermore, most bacteria used in bioreactors only thrive within a certain temperature range. Since these ones appear to be derived from a wild strain in the Chesapeake bay, this may not be the case, but it really depends on the genetic tinkering that was done (mostly if the genes were taken and put into a new host strain or if the original host is still used).

Aside from that, it comes down to what I vaguely recall from my ecology classes; the rule of 10%. Basically (again, going from memory here, so I may be totally off), if you take a species and put it in a new environment, there's a 10% chance it will survive. Of those that survive, there's a 10% chance that a breeding population will become established. 10% of the breeding populations will become problematic. All in all, I'm not really worried.

Can the monitor you're working on right now even display a 12MP image(4000x3000)? Sure there will be higher and higher pixel counts available, but at some point they're simply superfluous outside of specialist niches.

Rather than a computer analogy, here's a car one. The speed your car travels is highly dependent on external factors. Legal issues aside, there aren't many circumstances in a typical daily commute that really warrant speeds in excess of 100mph. Road conditions and the need for safety start to impose diminishing returns on increases in speed. So why do you go after a car with an LS9 (6.2L supercharged V8) under the hood when a B16 (common 4 cylinder Honda engine) is more than sufficient for 99% of your needs?

Right now, 12MP is more than sufficient for a typical consumer - in fact, it's arguably excessive already. Their displays can't handle images at that resolution, storage media and inbox space fill up too quickly, and all that excess resolution is mostly noise anyway (joe average doesn't understand iso settings). Granted, niche areas exist where higher resolutions are necessary (in the analogy they'd be drag racers), but diminishing returns set in at some point and the typical consumer will be much better served by something simpler.

Momofuku Ando, the guy who invented ramen noodles thought something similar: "Peace will come to the world when the people have enough to eat."

The problem is, as you provide for peoples' needs, they start to bicker about pettier and pettier things. For instance, look at the violence that breaks out between fans of opposing sport teams.

Are photographers real artists? An artist with a brush creates real art; a photographer merely finds it lying around.

Seriously tho, The camera is a tool, just as photoshop and gimp are tools. Some artists know how to use a tool well, others don't. People who know and appreciate art are quickly able to separate the real artists from the filter-happy pretenders. Just as the real audiophiles will quickly be able to distinguish between a great musical performance and over-compressed auto-tuned garbage.

Short answer: probably not. Telomere shortening does occur, and it does limit the number of divisions that certain cells can undergo. However, as I understand it, it's not the primary cause of aging symptoms. In fact, the lengthening of telomeres is associated with many kinds of cancer - not eternal youth. One gene that may be at least partially responsible for aging is Klotho. Experiments have been done in mice doing both knockdown expression and upregulation of the gene. Also, this is the gene that was making Snake age prematurely in MGS4.

Do what half of my graduating cohort is doing: go back to school for a PhD. You'll have all of your federal loans put back in forbearance and your living expenses covered by a stipend. It's not the glamorous high-paying job you dreamed about, but it'll give you a safe haven while this financial storm blows over. Better yet, you'll be even more competitive for jobs, and able to demand a higher salary a few years from now (in theory at least).