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Comment Re:Not really (Score 1) 756

Honestly, reading under those conditions isn't really all that tough and I have no problem using my phone on the road, other than the small screen size. The point I was trying to make is that although netbooks are nice, they definitely aren't the be all and end all of portable computing. It's nice to see more options appearing.

Comment Re:Not really (Score 3, Interesting) 756

Y'know, I love my netbook, but there are some times that it really just doesn't do it for me. Like in a yaris, at night, while someone else is driving down a gravel road. The position you're in is uncomfortable. The keyboard is awkward. The trackpad is tiny and useless when you're bouncing around.

It's not always how much something does, it's how easy it is to use. I'm going to be watching this very closely.

Comment Re:What is the point? (Score 1) 1713

Yes, that's exactly what I did, too, but I'm willing to put up with the warts.

I wouldn't say that it'll be "easier" to get it going, though. The linux "community" is way to ADD-addled to focus on polishing something for any length of time.

The warts must be removed if something is going to be even vaguely viable. People won't adapt to something new without a damn good reason, and even though us geeks think so, viruses are NOT enough of a concern to Joe Public.
Biotech

"Normal" Prions May Protect Myelin 81

thomst writes "Nature Neuroscience just published an online article about the function of 'normal' prions in protecting myelin, the substance that sheathes and protects sensory and motor nerves. The international study (which has 11 authors) concluded that 'normal' (i.e., not mis-folded) prions may form a protective coat around myelin. The researchers found that Prnp -/- mice (mice with the gene for prions knocked out) consistently developed progressive demyelination, inevitably leading to persistent polyneuropathy by 60 weeks of age. Their data suggest that damage to myelin sheaths cause normal prions to cleave, and the resulting prion fragments activate Schwann cells, which are known to play a part in myelin repair. This research might eventually lead to possible treatments for progressive polyneuropathies in humans, including those mediated by Creutzfeldt-Jakob disease, multiple sclerosis, Alzheimer's, and even diabetes."

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