Probably not. Different viruses have different protein coats, and antibodies are very specific on what they attach themselves onto. Should we manage to find a way around this problem (creating specific antibodies for other virions), the next problem would be an even bigger one. Common cold is a positive sense ssRNA virus meaning that its genome is a single stranded piece (or pieces, can't remember) of RNA that functions directly as mRNA for making proteins. Herpes viruses are dsDNA viruses meaning that their genomes consist of a piece of dual stranded DNA. This "virus-crushing machinery (TFA used this word)" that the antibody activates would probably be of no use towards this kind of molecules. It might be of useful for the +ssRNA hepatitis viruses (but HVB is dsDNA virus) and HIV (AIDS IS NOT A VIRUS, BUT A STATE) which genome is also +ssRNA molecule, but I doubt this very much. It all depends on the mode of action of this "virus-crushing machinery". I'm guessing it means RNAse (stuff that breaks RNA molecules). At least HIV would probably be safe, because it becomes dsDNA (and part of your genome) very quickly once it has entered a cell.