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Comment: Re:Yeah, like that'll work (Score 1) 112

So you're planning on putting lots of these expensive, hardened systems anywhere there 'might' be a disaster? Makes absolutely no sense.

If a disaster is that likely in a given locale, it would make much more sense to build your bunker - and fill it with thousands of pounds of supplies instead of letting your tiny toy copter bring you a couple of cans of beer and some joints. Or do what the Air Force does, put the stuff in pallets, attach a parachute and airlift it to where it's needed.

This is like trying to download Wikipedia on a 300 baud modem line.

Comment: Re:antibiotics are bad (Score 1) 221

Well, that definition would also apply to your classical 'antibiotic'. It appears from the Wikipedia site that Triclosan is not a generic antimicrobial in that it won't affect viruses, protozoa or Scientologists.

At in-use concentrations, triclosan acts as a biocide, with multiple cytoplasmic and membrane targets.[16] At lower concentrations, however, triclosan appears bacteriostatic and is seen to target bacteria mainly by inhibiting fatty acid synthesis. Triclosan binds to bacterial enoyl-acyl carrier protein reductase enzyme (ENR), which is encoded by the gene FabI. This binding increases the enzyme's affinity for nicotinamide adenine dinucleotide (NAD+). This results in the formation of a stable ternary complex of ENR-NAD+-triclosan, which is unable to participate in fatty acid synthesis. Fatty acids are necessary for reproducing and building cell membranes. Humans do not have an ENR enzyme, and thus are not affected. Some bacterial species can develop low-level resistance to triclosan at its lower bacteriostatic concentrations because of FabI mutations, which results in a decrease of triclosan's effect on ENR-NAD+ binding, as shown in Escherichia coli and Staphylococcus aureus.[17][18] Another way for these bacteria to gain low-level resistance to triclosan is to overexpress FabI.[19] Some bacteria have innate resistance to triclosan at low, bacteriostatic levels, such as Pseudomonas aeruginosa, which possesses multi-drug efflux pumps that "pump" triclosan out of the cell.[20] Other bacteria, such as some of the Bacillus genus, have alternative FabI genes (FabK) to which triclosan does not bind and hence are less susceptible.

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